Differences in working memory capacity is very limited, and a large legacy. The objectives to better understand the genetic basis for accurate working memory, research is mainly focused on adding a functional gene involved in the metabolism of the neurotransmitter dopamine. Recently, the study of genes related to other systems, such as the neurotransmitter acetylcholine.
The potential importance of DDR2 memory is to upgrading your memory, which codes for the nicotinic acetylcholine receptor discussed in working memory, but empirical studies have provided mixed results. However, the pharmaceutical studies of rodents and humans has shown that the nicotinic agonist effect on cognitive function through dopamine. It is therefore assumed that such interaction can be achieved for DDR2 memory using with the regular using. Examine this hypothesis, we genotyped healthy subjects and three functional polymorphisms of the DDR2 memory receptor memory. The theme, this is how to working memory tasks that systematically varied the memory load.

It is for example 2GB DDR2 memory that before found a significant interaction between another blocks, and all three parts of memory of working memory capacity. This effect is only apparent higher level of working memory load. This is the first evidence of genetic two neurotransmitter systems that affect cognitive function. Discuss the results, having regard to the consequences of working memory theory and clinical significance of drug treatment for the memory.